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Laser Vein Removal Philadelphia
Laser Vein Removal in Philadelphia. Treatment of varicose veins by laser coagulation. There is truth in my feet.
Varicose veins, or varicose veins, in everyday life is often referred to as "the disease of tired legs." But in fact, the factors causing it are much more complicated.
And the disease itself is by no means just a harmless cosmetic defect. Varicose veins of the lower extremities leads to the occurrence of chronic venous insufficiency - a condition, the final result of which may be the occurrence of a venous trophic ulcer.
Of course, in the overwhelming majority of cases we are dealing with varicose veins of moderate severity, which is now treated effectively and practically without relapse. But first things first.
Why is varicose veins dangerous?
It is difficult to say how common varicose veins disease is: many patients consider it a cosmetic defect and do not go to a doctor. According to Western scientists, at least a quarter of the population in the United States and Europe suffers from it. In Russia, the pathology was recorded in more than 30 million people . At the same time, according to some studies, no more than 18% know about their disease, and no more than 8% receive treatment .
Varicose veins of the lower extremities is a disease in which the structure of the vein wall changes. The vessels become longer, convoluted, in the areas of thinning of the walls, the lumen expands, forming nodes.
Varicose veins is a disease with a hereditary predisposition. The probability of its occurrence in those in whose family no one has suffered from venous pathology is no more than 20% . If one of the parents is sick, the risk increases: for men - up to 25%, for women - up to 62%. In the presence of the disease in both parents, the probability of occurrence of varicose veins in the offspring is 90%.
With varicose veins, the ratio of the two main structural proteins of the venous walls is disturbed: collagen and elastin. If collagen forms stiffness, then elastin, as its name suggests, is responsible for elasticity - the ability of the vein wall to return to its original state. In patients with varicose veins, the amount of this protein in the venous wall is reduced . Collagen itself also changes: instead of the prevailing type III collagen, which is responsible for elasticity, the content of type I collagen - rigid, retaining residual deformation, increases. In addition, the number of smooth muscle cells , which regulate the vessel lumen, also changes, and the ability to interact between them is impaired . These pathological changes are hereditary. Further, external factors come into play:
- long-term static loads - the need for motionless standing or sitting;
- pregnancy and childbirth.
- Some experts point to chronic constipation, tight clothing that increases intra-abdominal pressure, and high heels that interfere with the normal functioning of the muscle pump in the legs as predisposing factors.
Individually or in combination, these factors increase the pressure within the venous system of the lower extremities. The altered venous wall ceases to "hold" pressure, the lumen of the vein expands. As a result of the expansion of the lumen of the vein, the valves, which provide blood flow in only one direction, stop working. Reflux occurs - backflow. The pressure inside the affected vein rises even more, and the vicious circle is closed.
Increased venous pressure in combination with a change in the structure of the wall over time triggers an inflammatory reaction - at first only on the surface of the valves and the inner wall of the vessels. Gradually, inflammatory proteins and blood cells begin to "seep" through the damaged venous wall into the surrounding tissue. There they are destroyed, releasing active substances - inflammatory mediators. They damage the surrounding cells and attract lymphocytes, whose function is to remove damaged tissue. As a result, edema, induration (induration) of the skin of the legs, hyperpigmentation develop. These processes can cause venous trophic ulcers. Prolonged inflammation of the venous wall also increases blood clotting. In combination with venous congestion, this leads to the fact that blood clots begin to form in the varicose vein. Thrombophlebitis occurs - another dangerous complication of varicose veins.
The manifestations of varicose veins are divided into objective and subjective. Subjective symptoms are patient complaints about:
- heaviness in the legs;
- rapid fatigue;
- paresthesias - unpleasant sensations of "goose bumps", changes in sensitivity;
- burning sensation in the muscles;
- pain in the legs, the characteristic feature of which is that the intensity decreases after a walk;
- swelling in the evening;
- restless legs syndrome - a condition in which discomfort in the legs prevents sleep;
- night cramps in the lower limbs.
The combination of these symptoms and their severity are individual and do not always correlate with changes in the venous wall and surrounding tissues.